Genome-Wide Association Study Evaluating Lp-PLA2 Mass and Activity at Baseline and Following Rosuvastatin Therapy

نویسندگان

  • Audrey Y. Chu
  • Franco Guilianini
  • Harald Grallert
  • Josée Dupuis
  • Christie M. Ballantyne
  • Bryan J. Barratt
  • Daniel I. Chasman
  • Paul M. Ridker
چکیده

Center for Cardiovascular Disease Prevention, the Division of Preventive Medicine, JUPITER Trial Coordinating Center, Division of Genetics, Division of Cardiology, Brigham and Women’s Hospital, Boston, MA; Helmholtz Zentrum München, German Research Center for Environmental Health, Research Unit of Molecular Epidemiology, Neuherberg, Germany; National Heart, Lung, and Blood Institute’s & Boston University's Framingham Heart Study, Framingham; Dept of Biostatistics, Boston University School of Public Health, Boston, MA; Division of Atherosclerosis & Vascular Medicine, Dept of Medicine, Baylor College of Medicine, Methodist DeBaakey Heart and Vascular Center, Houston, TX; Personalised Healthcare & Biomarkers, AstraZeneca Research & Development, Alderley Park, UK; Global Epidemiology, AstraZeneca Research and Development, Mölndal; Unit of Occupational and Environmental Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden; Harvard Medical School, Boston, MA †contributed equally

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Genome-wide association study evaluating lipoprotein-associated phospholipase A2 mass and activity at baseline and after rosuvastatin therapy.

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تاریخ انتشار 2012